HSPA8 Activates Wnt/β‐Catenin Signaling to Facilitate BRAF V600E Colorectal Cancer Progression by CMA‐Mediated CAV1 Degradation - Li - Advanced Science - Wiley Online Library

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Regulation of signal transduction pathways in colorectal cancer: implications for therapeutic resistance [PeerJ]

Emerging agents that target signaling pathways to eradicate colorectal cancer stem cells - Silva - 2021 - Cancer Communications - Wiley Online Library

PDF] How the BRAF V600E Mutation Defines a Distinct Subgroup of Colorectal Cancer: Molecular and Clinical Implications

Wnt/β-catenin signaling in colorectal cancer: Is therapeutic targeting even possible? - ScienceDirect

BRAFV600E drives dedifferentiation in small intestinal and colonic organoids and cooperates with mutant p53 and Apc loss in transformation

Unresponsiveness of colon cancer to BRAF(V600E) inhibition through feedback activation of EGFR

LINC00665 activates Wnt/β-catenin signaling pathway to facilitate tumor progression of colorectal cancer via upregulating CTNNB1 - ScienceDirect

Wnt/β-catenin signaling and p68 conjointly regulate CHIP in colorectal carcinoma - ScienceDirect

Molecular targeted therapy of BRAF-mutant colorectal cancer - Michel Ducreux, Ali Chamseddine, Pierre Laurent-Puig, Cristina Smolenschi, Antoine Hollebecque, Peggy Dartigues, Emmanuelle Samallin, Valérie Boige, David Malka, Maximiliano Gelli, 2019

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BRAF-Mutated Advanced Colorectal Cancer: A Rapidly Changing Therapeutic Landscape